too much iodine causes thyroid issues too and you might still become hypothyroid as the gland stops processing it properly ... it gets damaged by too much iodine.
give your iodine intake days a gap or reduce the amount. use uniodised salt in case you are using iodine everyday.
But is it too much iodine causing the problem? Or too little selenium and co-factors causing it?
ichealer.com/iodine-deficiency-and-hashimotos/
Hydrogen peroxide is a byproduct of oxidative phosphorylation. This is a process your body uses to produce energy in the form of ATP. The production of hydrogen peroxide is critical to the oxidation process of iodine! Hydrogen peroxide and TPO help to oxidize iodide to form iodine (Brownstein, 2014). Adequate hydrogen peroxide production is essential for facilitating the oxidation of iodide (Brownstein, 2014). Without adequate levels, iodine will not be formed from iodide. However, if the production and utilization of hydrogen peroxide is not closely maintained, it can damage the tissue and cause autoimmune thyroid disorders such as Hashimoto’s disease.The immune system has antioxidant mechanisms to keep from hydrogen peroxide from harming tissues. These include selenium-containing enzyme glutathione peroxidase which functions to keep hydrogen peroxide reduced to water after it oxidizes to iodide. A deficiency in selenium or precursors to glutathione (glycine, glutamine, cysteine) can impair this defense mechanism. Once iodine is formed, it undergoes organification and becomes part of cholesterol, lipids and proteins.
When you ingest RDA levels of iodine, it can lead to iodine deficiency. There is not enough iodine to produce iodinated lipids, such as δ-iodolactone and others that puts a brake in the pathway to oxidize iodide. This may result in a temporary production of TOO MUCH hydrogen peroxide. This excessive hydrogen peroxide can damage the enzyme TPO.
..you need enough iodine to produce iodo-lipids such as δ-iodolactone. You also need to stimulate the NADPH oxidase pathway that is integral to energy production. That would require providing the cofactors Vitamin B2 (riboflavin) and B3 (niacin) that are cofactors of the NADPH oxidase system that will not only stimulate the mitochondria to product more energy but also provide thyroid cells with enough hydrogen peroxide to properly oxidize iodide.
This is why iodine supplementation is not as simple. Ive known many to supplement without selenium, get thyroid problems and then reduce the iodine when they really should have been ensuring they had enough selenium to offset the natural oxidative stress that occurs when the body processes the thyroid hormones. Given that our environment, food supply, water supply, the fire retardant chemicals in furniture and the depleted soils, how likely are people to be getting too much iodine or selenium for that matter?
Small Pox
